Strategies to Slow Down Aging: The Power of Frankincense
According to researchers, viable anti-aging strategies may include:
- consuming antioxidants, especially from fruits and veggies
- caloric restriction to quench cell-damaging free radicals
- a balanced diet of proteins, good fats, and complex carbohydrates
- stress reduction or meditation (to avoid the poison of the hormone cortisol)
- good sleep
- regular exercise (and if you're exercising a lot, be sure to intercept those extra free radicals produced with antioxidants)
- avoidance of environmental and food-borne toxins
- supplementation of nutrients that decline with aging (such as CoQ10)
- drinking lots of water
- reducing inflammation
Without inflammation present in the body, there is no way that cholesterol could accumulate in the walls of our blood vessels and cause heart disease and strokes. Without inflammation, cholesterol would move freely throughout the body as nature intended. It is inflammation that causes cholesterol to become trapped.
Reputable scientists worldwide are finding that inflammation may be the root cause of almost every major disease out there, from arteriosclerosis to autoimmune diseases to ulcerative colitis to allergies to cancers of all kinds.
When one looks deeper and deeper through an abundance of scientific research into the nature of many debilitating and killer diseases, the search for anti-aging strategies almost invariably points to the importance of reducing inflammation, which in turn commonly points to frankincense as an important tool in inhibiting inflammation.
FRANKINCENSE: COULD IT REALLY BE A "FOUNTAIN OF YOUTH"?
Science now recognizes that frankincense, an aromatic resin obtained from trees of the genus Boswellia, possesses therapeutic anti-inflammatory properties. (Boswellia is called Salai guggul in ancient Ayurvedic medicine, where guggul means "one that protects against diseases.")
Along with the anti-aging strategies listed above, below are numerous scientific research articles validating the profound effects of Boswellia on your body, helping to keep it young by nipping inflammation in the bud. It's this simple: add Frankincense Essential Oil to your sacred medicine bag.
Couple this with ample scientific evidence that the colon is a superior delivery system for therapeutic agents, and I suggest we have a powerful anti-aging essential oil to use.
Use 4-20 drops of frankincense in the colon as often as once a day. Deliver it via a suppository, colon implant, or coffee enema.
- Buy Frankincense and Myrrh Made-for-Enema Goat Milk Soap
- Buy Kristina's book, Ten Days to Optimal Health,
for support with an anti-inflammatory diet
FRANKINCENSE RESEARCH TO EXPLORE:
- RESIN OF BOSWELLIA HAS ANTI-INFLAMMATORY, ANTI-ARTHRITIC AND ANALGESIC PROPERTIES
Natural anti-inflammatory agents for pain relief.
- FRANKINCENSE INHIBITS ATHEROSCLEROSIS
Anti-inflammatory and anti-atherogenic effects of the NF-kappaB inhibitor acetyl-11-keto-beta-boswellic acid in LPS-challenged ApoE-/- mice.
"The inhibition of NF-kappaB activity by plant resins from species of the Boswellia family might represent an alternative for classical medicine treatments for chronic inflammatory diseases such as atherosclerosis."
- FRANKINCENSE INHIBITS COLORECTAL CANCER
Boswellic acid inhibits growth and metastasis of human colorectal cancer in orthotopic mouse model by downregulating inflammatory, proliferative, invasive and angiogenic biomarkers.
"Our findings suggest that this boswellic acid analog can inhibit the growth and metastasis of human CRC in vivo through downregulation of cancer-associated biomarkers."
- FRANKINCENSE INHIBITS INFLAMMATORY BOWEL DISEASE
Acetyl-11-keto-beta-boswellic acid, a constituent of an herbal medicine from Boswellia serrata resin, attenuates experimental ileitis**.
"Oral therapy with the Boswellia extract or AKBA significantly reduces macroscopic and microcirculatory inflammatory features normally associated with indomethacin administration, indicating that the anti-inflammatory actions of the Boswellia extract in IBD [inflammatory bowl disease] may be due in part to boswellic acids such as AKBA."
** Ileitis, or inflammation of the ileum (a portion of the small intestine), is often caused by Crohn's disease. However, ileitis may be caused by a wide variety of other diseases.
- BOSWELLIC ACIDS SHOW ANTI-INFLAMMATORY AND ANTI-TUMORIC PROPERTIES
Inhibition of microsomal prostaglandin E2 synthase-1 as a molecular basis for the anti-inflammatory actions of boswellic acids from frankincense.
"Frankincense, the gum resin derived from Boswellia species, showed anti-inflammatory efficacy in animal models and in pilot clinical studies... There is [also] accumulating evidence for... anti-tumorigenic effects of BAs. Pilot clinical studies have suggested some efficacy of frankincense preparations in the treatment of osteoarthritis (OA), rheumatoid arthritis (RA), inflammatory bowel diseases, asthma and cancer."
- FRANKINCENSE INHIBITS VARIOUS CHRONIC INFLAMMATORY DISEASES AND CANCER
Boswellic acids: biological actions and molecular targets.
"Gum resin extracts of Boswellia species have been traditionally applied in folk medicine for centuries to treat various chronic inflammatory diseases, and experimental data from animal models and studies with human subjects confirmed the potential of these extracts for the treatment of not only inflammation but also of cancer."
- FRANKINCENSE IS POWERFUL ANTI-INFLAMMATORY AND ANTI-ARTHRITIC AGENT; AIDS RECOVERING ARTICULAR CARTILAGE DAMAGE
Cellular and molecular mechanisms of anti-inflammatory effect of Aflapin: a novel Boswellia serrata extract.
"BE-30 (5-Loxin) and Aflapin [which contains B. serrata extract enriched in AKBA and non-volatile oil portion of B. serrata gum resin], exhibit powerful anti-inflammatory efficacy and anti-arthritic potential. In particular, in comparison with BE-30, Aflapin provides more potential benefits in recovering articular cartilage damage or protection from proteolytic degradation due to inflammatory insult in arthritis such as osteoarthritis or rheumatoid arthritis."
- FRANKINCENSE INHIBITS RHEUMATOID ARTHRITIS, CHRONIC COLITIS, ULCERATIVE COLITIS, CROHN'S DISEASE, BRONCHIAL ASTHMA, AND PERITUMORAL BRAIN EDEMAS
Boswellic acids (components of frankincense) as the active principle in treatment of chronic inflammatory diseases.
"Preparations from the gum resin of Boswellia serrata have been used as a traditional remedy in Ayurvedic medicine in India for the treatment of inflammatory diseases. Compounds from the gum with genuine anti-inflammatory effects are pentacyclic triterpenes of the boswellic acid type. Boswellic acids inhibit the leukotriene biosynthesis in neutrophilic granulocytes by a non-redox, noncompetitive inhibition of 5-lipoxygenase. The effect is triggered by boswellic acids binding to the enzyme. Moreover certain boswellic acids have been described to inhibit elastase in leukocytes, to inhibit proliferation, induce apoptosis and to inhibit topoisomerases of leukoma and glioma cell lines. A series of chronic inflammatory diseases are thought to be perpetuated by leukotrienes. In clinical trials promising results were observed in patients with rheumatoid arthritis, chronic colitis, ulcerative colitis, Crohn's disease, bronchial asthma, and peritumoral brain edemas."
- FRANKINCENSE INHIBITS ASTHMA, CHRONIC OBSTRUCTIVE PULMONARY DISEASE, EMPHYSEMA, REPERFUSION INJURY, PSORIASIS, RHEUMATOID ARTHRITIS, CROHN'S DISEASE
Identification of human cathepsin G as a functional target of Boswellic acids from the anti-inflammatory remedy frankincense.
"[Cathepsin G] catG is a functional and pharmacologically relevant target of BAs, and interference with catG could explain some of the anti-inflammatory properties of frankincense...CatG, a neutral serine protease, is mainly expressed in neutrophils, stored in azurophilic granules, and released upon degranulation. After release into the plasma, it cleaves extracellular matrix proteins, including laminin, proteoglycans, collagen, fibronectin, and elastin, implying a role in local destruction of connective tissue at sites of injury. CatG also processes chemokines, functioning as chemoattractant for T cells and other leukocytes, and modulates integrin clustering on neutrophils. Moreover, catG stimulates platelets via the protease-activated receptor-4 for aggregation and secretion and acts as chemotactic agonist for the formyl peptide receptor on phagocytic leukocytes. Accordingly, catG inhibitors have been proposed to exhibit potential in treating certain inflammatory disorders such as asthma, chronic obstructive pulmonary disease, emphysema, reperfusion injury, psoriasis, and rheumatoid arthritis."